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Karum (Clopidogrel)

Karum (brand name Karum) is an antiplatelet medicine. This means it reduces the risk of blood clots forming. Normally, when there is a cut or break in a small blood vessel, a blood clot forms to plug the hole until the blood vessel heals. Small cells in the blood called platelets cause the blood to clot. When a platelet detects a damaged area of a blood vessel, it produces a chemical that attracts other platelets and makes them stick together to form a blood clot. Karum reduces the ability of the platelets to stick together and reduces the risk of clots forming. This protects you from having a stroke or heart attack.

Other names for this medication:
Clopidogrel, Plavix, Anclog, Anlet, Antiplar, Artevil, Clavix, Clopicard, Clopilet, Clopivas, Deplatt, Diloxol, Duoplavin, Expansia, Farcet, Globel, Grepid, Iscover, Karum, Klopidogrel, Nabratin, Narutis, Nefazan, Noklot, Pidocar, Pidogrel, Pladex, Plagerine, Planor, Tisten, Troken, Trombex, Vaclo, Zyllt,

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Citol, Clofen, Levoxin, Doxin, Lovequin, Neozentius, Amoxibeta, Delak, Bexinor, Primazole, Circuvit, Difluzol, Anafranil, Hydertan, Zoxil, Zocef, Virestat, Dulane, Lindan, Esram, Megapen, Nisamox, Exten, Levoquin, Talosin, Aerodan, Pantoprazole, Amoxipen, Amox, Clavulox, Lanx, Lutamidal, Turimycin, Oratil, Stafcure, Sawacillin, Gabictal, Dineurin,

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Foreign bodies in the bronchi and their sequelae in young persons Proteolytic degradation of neuropeptide Y (NPY) from head to toe: Identification of novel NPY-cleaving peptidases and potential drug interactions in CNS and Periphery. To begin to investigate how pathogenic leptospires affect endothelial cells, we compared the responses of two endothelial cell lines to infection by pathogenic versus non-pathogenic leptospires. elegans: a framework for simulation and visualization of the neuromuscular system in a 3D physical environment.

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Two alleles, m1 and m2, of the mid-region of the vacA gene have been described, and the m2 cytotoxin always has been described as inactive in the in vitro HeLa cell assay. The resolved flux distributions demonstrate that the glyoxylate shunt pathway is constantly active and the flux through gluconeogenesis varies depending on strain and phase. The effects of hypo-(260 mOsm) and hypertonic (350 mOsm) osmolarity on the activation and inactivation kinetics of VGSCs on TRGN were evaluated, compared with the normal intracellular environment. Prolonged exposure to tyrosine kinase inhibitors or early use of everolimus in metastatic renal cell carcinoma: are the two options alike?

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There were no significant predictors of child-reported PTSD-specific symptoms, although PTSD symptoms did decrease significantly over the course of treatment and maintain 1year after treatment. Recombinant human growth hormone exacerbates chronic puromycin aminonucleoside nephropathy in rats. Effects of corticosteroid, montelukast and iNOS inhibition on distal lung with chronic inflammation.

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Normal genetic variation of the human foot: part 1: the paradox of normal anatomical alignment in an evolutionary epigenetic context. To evaluate changes in anorectal symptoms before and after pelvic organ prolapse (POP) surgery, using laparoscopic sacrocolpoperineopexy.

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